Actinomyces viscosus

A. viscosus is Gram-positive, facultatively anaerobic, rod-shaped, and filamentous. It grows slowly on nonselective media, forming gray and white colonies. ==Pathogenesis==

A. viscosus causes periodontal disease in animals and has been isolated from human dental calculus and root surface caries, as well as the oral cavity of hamsters and actinomycotic lesions in swine, cats, and dogs. Furthermore, it has been shown to cause endocarditis in humans. A. viscosus has also been known to cause lung infections, but only in very few cases. Infections are treatable with penicillin for three-week therapies. == Diagnosis ==

A. viscosus infection symptoms are indistinguishable from Actinomyces israelii infection symptoms or Actinomyces bovis infection symptoms. == Treatment ==

Multiple-week antibiotic therapies have cured actinomycotic infections caused by A. viscosus in every recorded case. Therapies include treatment with penicillin, sulfadimethoxine, flucloxacillin, clindamycin, tetracycline, and ticarcillin. A. viscosus is usually resistant to vancomycin, metronidazole, cefalexin, and dicloxacillin. Treatments last at least three weeks, with some exceptions. Although A. viscosus is difficult to distinguish from other closely related actinomycetes, the general determination of being an actinomycete is sufficient for treatment of infections. ==References==