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Ecoimmunology

Ecoimmunology or Ecological Immunology is the study of the causes and consequences of variation in immunity. The field of ecoimmunology seeks to give an ultimate perspective for proximate mechanisms of immunology. This approach places immunology in evolutionary and ecological contexts across all levels of biological organization.

History
Ecological Immunology is a discipline that uses ecological perspectives to understand variation in immune function. Specifically, to explain how abiotic and biotic factors influence the variation in immune function. Ecoimmunology is an integrative field that combines approaches from evolutionary biology, ecology, neurobiology, and endocrinology. was a response to Hamilton and Zuk's famous paper on the handicap hypothesis for sexually selected traits. Folstad and Karter proposed the immunocompetence handicap hypothesis, whereby testosterone acts as a mediator of immunosuppression and thus keeps sexually selected traits honest. Although there is only moderate observational or experimental evidence supporting this claim up until now, the paper itself was one of the first links to be made suggesting a cost to immunity requiring trade-offs between it and other physiological processes. More recently, ecoimmunology has been the theme of three special issues in peer-reviewed journals, in Philosophical Transactions of the Royal Society B, in Functional Ecology, and in Physiological and Biochemical Zoology (see External links). == Known factors that influence immune variation ==
Known factors that influence immune variation
Intraspecific constraints Organisms allocate energy between competing processes including self-maintenance, reproduction, or growth. Energy availability is limited, and the resources used for one of the competing metabolic tasks (i.e., growth, immune response) cannot be directed towards another. The cost of immunity is central to the understanding of ecoimmunology. Natural selection should favor the optimal immune response that maximizes total lifetime reproductive output. The costs of immunity to parasites occur at the individual and the evolutionary scale. Innate versus acquired One axis on which these trade-offs occur is the trade-off between innate and acquired immunity. McDade applies a framework that considers three ecological factors that shape life-history trade-offs. The framework suggests that environments with high extrinsic mortality should favor innate immunity or short-term immunity while low extrinsic mortality should allow for a longer time horizon in order to invest in acquired or long-term immunity. In Tsimane children, a 49% reduction in growth was observed in children with mild immune activation. Body size Body size affects the extent to which an organism is exposed to parasites as well as limitations on how organisms can mount an immune response. Reproduction Physiological and behavioral changes during reproduction are known to influence the immune system. Trade-offs occur between bodily maintenance (which includes immune function) and reproduction, as metabolic energy expenditure is increased during pregnancy and lactation. The reproductive system is unique in that its function is to produce offspring while the immune system provides internal protection. Both systems are regulated by chemical signals in response to environmental stimuli and rely on interactions between both systems in order for each to function properly. however it is unclear if changes in the immune system are causing this as few studies include measures for both immunity and parasitism. A study of wild red deer on the Isle of Rum, off the coast of Scotland, found that reproducing females had lower antibody levels and higher parasite counts. Testosterone The Immunocompetence Handicap Hypothesis and similar theories propose that testosterone mediates a trade-off between longevity and reproductive effort in males, prioritizing investment in secondary sexual characteristics such as sexually dimorphic muscle mass. Energetically expensive secondary sexual characteristics, such as skeletal muscle mass, have been shown to predict a relationship between testosterone levels and reproductive effort. Human males experience muscle mass deterioration during times of immunological and nutritional stress. and HIV. A different theoretical model has been proposed for testosterone variability as phenotypic plasticity taking into account behavioral and environmental impacts as well as the role of immune activation on testosterone levels. Activation of the hypothalamic-pituitary adrenal (HPA) axis is one of the main mechanisms by which the immune system interacts with stress. In animal studies, stressors such as social disruption and restraint stress active HPA axis in mice and nasal shedding in bovids. In humans, stress is a predictor of recurrences of herpes simplex virus outbreaks and Epstein-Barr virus. Interactions with parasites Host feeding behavior Parasite-altered feeding behaviors have been observed in several species. Most studies conclude that there is a fitness benefit of altering host feeding behavior to either the host or the parasite. The species S.littoralis caterpillar when infected with nucleopolyhedrovirus will self-select a protein-rich diet, which increases its probability of survival. Parasite manipulation Selection is expected to favor parasite manipulation of the host when the host's behavior creates a suboptimal environment for the parasite's fitness. An application of coevolutionary theory would predict sophisticated manipulations of host behavior when host-specificity is high. This phenomenon has been observed in several species, with the most prominent examples including the ingestion of whole leaves by primate species to reduce nematode infections and the ingestion of secondary plant metabolites by caterpillars and bumblebees. In social insects, behaviors that reduce colony-level parasite loads are termed "social immunity". An example of this, Apis mellifera incorporate plant resins in their nest building as this can reduce the chronic elevation of an immune response at the individual level. Immune costs are often seen when organisms are in stressful environments such as experiencing nutritional stress. In animal models, fruit flies that were selected for parasitoid resistance showed reduced larval competitiveness only when they were subject to food limitations. Leptin has been proposed as a mediator of energetic trade-offs, as a potential provider of signal for current energy availability. Microbiome Rapid changes in the gut microbiome occurred during human evolution Because the microbiome is influenced by the host environment, researchers believe that it played a role in facilitating human adaptation to novel environments facilitated through periods of climate change and migration. For instance, commensal microbes influence the host's ability to survive pathogenic exposures through several mechanisms including inter-microbial competition and interaction with the immune system. Population genetics Population genetic characteristics such as population size, mutation frequency, and selective processes are important host-parasite co-evolutionary dynamics and therefore influence the evolution of different aspects of the immune system. Pathogen-driven selection has been supported in allele frequency studies including MHC I and blood group antigens. Gene networks have also been correlated with specific pathogens including helminths. == Critiques ==
Critiques
Early studies in ecoimmunology tended to underestimate the complexities of parasite defenses, often relying on one or two immune metrics as an overall indication of anti-pathogen defense capabilities. To date, sampling wild populations have shown there is substantial inter-individual immune variation. Another source of criticism comes from the need for to develop assays that can be utilized across species and be accessible in multiple laboratories due to the fact that ecoimmunologists primarily study non-model organisms. == Evolutionary implications ==
Evolutionary implications
Ecoimmunology allows for the incorporation of more realistic details of variation in individual immune responses in a population. New research has demonstrated that individual variation in infectiousness follows a highly skewed distribution, with very few individuals being highly infectious. Models that account for heterogeneity, predicted rare more rapidly-spreading epidemics and argued for the use of different types of public health interventions compared to models that assume a normal distribution of variation in infectiousness. Recent advances in theoretical modeling have allowed for the increased integration of within-organism processes (such as immune-mediated reduction in pathogen replication) and between organism processes (such as transmission). == Medical implications ==
Medical implications
One of the most influential contributions of ecoimmunology has been the concept of tolerance which incorporates the cost of infection into measures of immunity. The study of tolerance has implications in human biomedicine, wildlife ecology, and public health. A shift in focus to tolerance rather than eradication might provide fruitful avenues for treatments that reduce virulence rather than eliminating parasites. == See also ==
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