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Potato virus Y

Potato virus Y (PVY) is a plant virus of the family Potyviridae, and one of the most important plant viruses affecting potato production.

Hosts, strains, and symptoms
PVY belongs to the genus Potyvirus, the largest genus of plant viruses and possibly the most destructive one in potato crops. The genus includes more than 200 species that bring about significant losses in the agricultural arena. PVY infects many economically important plant species. These include potato (Solanum tuberosum), tobacco (Nicotiana tabacum), tomato (Solanum lycopersicum) and pepper (Capsicum spp.). The level of damage to crop is determined by the strain of PVY infecting the plants, the viral load, the time at which infection occurs as well as the tolerance the host possesses toward the virus. Resistance to PVY infection by hosts is low in many cases. Infection of a potato field with PVY may ultimately result in 10–100% loss in yield. Extensive biological, serological and molecular variability of PVY isolates makes the classification of isolates as particular strains particularly difficult. Occurrence of a variety of symptoms and the emergence of the necrotic PVYNTN has led to a search for more reliable classification tools than simple serological identification. Traditionally three chief strains of PVY are recognized: PVYC, PVYN and PVYO. PVYC, originally known as Potato Virus C, was the first to be recognized and was identified in the 1930s. PVYC induces hypersensitive responses in a wide range of potato cultivars. These reactions include the formation of mild mosaic patterns or stipple streak. Unlike the other strains of PVY, some PVYC strains are non-aphid transmissible. Previous studies by Visser et al. did not identify any of the local isolates as being PVYC but it has been reported to occur in potatoes in South Africa. A second strain of PVY is PVYN. This strain was described in tobacco plants growing close to potato plants. PVYN results in leaf necrosis and mild or even no damage to the tubers. The ordinary strain of PVY is denoted as PVYO. Infection of a potato plant with the PVYO strain results in mild tuber damage and does not cause leaf necrosis. Both PVYN and PVYO are aphid transmissible and occur in South Africa. In Europe these two strains have been shown to have recombined to form PVYNTN. The PVYNTN has been accredited with the ability to induce potato tuber necrotic ringspot disease (PTNRD). Tubers damaged by PTNRD become unmarketable and infection by PVYNTN thus results in a larger economic impact than infection by the other strains. ==Transmission==
Transmission
PVY may be transmitted to potato plants through grafting, plant sap inoculation and through aphid transmission. The most common manner of PVY infection of plant material in the field is through the aphid, and although aphids on their own can directly damage potato plants, it is their role as viral vectors which has the greatest economic impact. In cold climates aphids spend the winter either as wingless aphids giving birth to live young (viviparae) or as eggs. Hosts such as weeds and other crops serve as breeding grounds for these aphids and form a temporary area of colonization before the aphids migrate to the potato fields. The green peach aphid (Myzus persicae) has been found to be most effective in its role as viral vector, but others such as Aphis fabae, Aphis gossypii, Aphis nasturtii, Macrosiphum euphorbiae, Myzus (Nectarosiphon) certus, Myzus (Phorodon) humuli and Rhopalosiphum insertum are also strongly associated with viral transmission. The efficiencies of some of these aphids to function as PVY vectors were also established (Ragsdale et al., 2001) and were found to vary between the different species. In South Africa, Aphis fabae, Aphis gossypii and Aphis nasturtii are the most common and efficient PVY vectors found in the field. Because of this, all aphids present in and around potato fields must be considered as possible vectors and their numbers carefully monitored. Transmission of PVY by aphids occurs in a non-persistent, non-circulative manner which suggests a less intimate interaction between virion and vector than is the case of circulative virions. The fact that the virions are transmitted in a non-persistent fashion means that viral replication does not occur within the aphid vector and that, unless the aphid feeds on infected plants, it loses its ability to infect plants after two to three feedings. The virions attach to the aphid stylet in a matter of seconds and may remain infectious for four to seventeen hours. The distance over which the virions can be transmitted is limited due to the short period for which they remain infectious. The translated mRNA results in a polyprotein which is processed into mature proteins. Each polyprotein is then cleaved into ten different proteins which are believed to be multifunctional. These proteins, along with host proteins, assemble to form a replication complex. This complex performs negative-strand RNA synthesis, using the positive strand of viral RNA as a template. Once the additional RNA copies have been produced, they code for the synthesis of various proteins, as mentioned before, as well as coat proteins. These coat proteins will now enclose the newly formed genomes to give rise to new virions. It has been suggested that enclosure of the newly formed virions is initiated by the interaction of the coat proteins with the 5’terminus and that the coat protein is built up towards the 3’terminus. The entire process of viral replication occurs within the endoplasmic reticulum. These newly synthesized viral particles are subsequently transported through the plasmodesmata to adjacent plant cells via several assisting potyvirus proteins. Distribution of viruses within the plant occurs according to the source-sink relationship between maturing and growing tissues. Virus concentration throughout the plant is high and this greatly increases the chance of uptake by aphids. Infection of plants by potyviruses can be varied in the symptoms shown. Infection can include veinal necrosis, mosaic symptoms as well as leaf malformation (Boonham et al., 2002). Infected plants that do not show symptoms may have infected canopies and will yield lower quality products than their healthy counterparts. ==Potato – PVYNTN interaction==
Potato – PVYNTN interaction
Since PVYNTN causes great loss in potato production, the research of potato – potato virus YNTN interaction is important. Sensitive potato cultivars respond to PVYNTN inoculation with development of typical symptoms. On inoculated leaves 5 – 7 days after inoculation chlorotic and necrotic ringspots develop. As the virus spreads through the plant the systemic symptoms develop on uninoculated leaves. 10 days after inoculation wrinkles and mosaic chlorosis appear, leading to a palm tree appearance (leaf drop). The viral defense mechanisms of plants will primarily try to restrict the movement of the virus. In failing this, it may attempt to induce cell death in infected tissue, thereby preventing the spread of virions. Although the precise mechanism of disease induction by potyviruses in plants is unknown, it is known that these viruses cause a significant shutdown of host gene expression during viral replication. Physiological changes in potato plants as a response to PVYNTN infection were intensively studied. At early stages of infection, meaning first 12 hours, photosynthesis related genes, genes involved in perception, signalling and defence response were shown to be differentially expressed. A disruption in gene expression disrupts the normal cellular function of cells which could be the cause of the physical symptoms that the plant demonstrates. At the time of symptoms development, research on interaction between susceptible potato cultivar and PVYNTN showed changes in cytokinin level. In inoculated leaves showing symptoms modifications in chloroplast structure and size, lower chlorophyll levels and differential activity of soluble and ionically bound peroxidases were detected. At later stages of PVYNTN infection total protein concentration increased in sensitive potato cultivar while no such pronounced changes were observed in tolerant and moderately tolerant potato cultivars. Gene expression studies revealed changes in expression of genes for heat-shock proteins, catalase, β-1,3-glucanase and genes involved in photosynthesis. ==Molecular description==
Molecular description
Potyvirus virions consist of non-enveloped filamentous structures that are 680 – 900 nm in length and 11 to 15 nm in width. Morphologically the potyvirus capsid consists of approximately 2 000 copies of coat protein (CP). The positive sense genome contains a single extended open reading frame and acts directly as mRNA. The 144 nucleotide 5’-NTR is particularly rich in adenine residues and has very few guanine residues. Rather than a conventional cap structure, the 5’NTR is associated with a Viral genome linked protein (VPg) which is said to act as an enhancer of transcription. The IRES directs cap-independent translation through a mechanism similar to that used by eukaryotes. The extended open reading frame encodes for a 350 kDa polyprotein. This polyprotein is proteolytically processed by viral proteases (NIa, HC-Pro and P1) and undergoes co- and post-translational cleavage to yield several multi-functional proteins. These include the following: P1 (P1 Protein), HCPro (Helper Component Proteinase), P3 (P3 Protein), 6K1 (6-kDa Protein 1), CI (Cylindrical Inclusion), 6K2 (6-kDa Protein 2), VPg (Viral Protein genome-linked), NIaPro (Nuclear Inclusion Protein a, Proteinase domain), NIb (Nuclear Inclusion Protein b) and the CP (Coat Protein). ==Diagnosis and detection==
Diagnosis and detection
ELISA In the past, crops were inspected visually to determine whether or not they were disease free. Visual inspection was also used as a basis for seed certification. Determination of viral status through visual inspection is incredibly difficult as the symptoms may be masked or the infection latent. The efficiency to which the antigen binds to the solid phase is dependent on temperature, length of exposure as well as concentration. Nonspecifically bound components are less strongly bound than the specific bound ones. Detection is achieved either through the addition of an enzyme-coupled antibody or the addition and detection of a biotinylated antibody. In a system using an enzyme-coupled antibody the subsequent addition of an appropriate substrate results in the formation of a colour proportional to the amount of antigen. This may be overcome through the use of a biotin-avidin or biotin-streptavidin bridge. In this type of system biotin is coupled to the antibody. The biotin molecule has no influence on the working of the antibodies and is easily detectedusing avidin or streptavidin conjugated to a suitable enzyme. Streptavidin has an extremely high affinity for biotin which results in even a higher degree of specificity than a system in which the enzyme is coupled directly the antigen. To establish whether or not the antigen is present, a substrate specific for the enzyme used is added. The enzyme then converts the substrate to a coloured product and the colour intensity can be correlated to the amount of antibodies bound and thus the amount of antigen present. A DAS-ELISA has the advantage that it can increase the specificity of the ELISA and reduce the occurrence of non-specific binding. As a result, the DAS-ELISA principle is commonly employed in ELISA's for the detection of plant pathogens in plant sap without prior purification of the pathogen. The ELISA is considered to be a safe, inexpensive and rapid method for detection of plant viruses. The inexpensive nature and relative simplicity thereof allows for it to be used as a workhorse within the agricultural sector and is used to screen thousands of samples per year. Unfortunately ELISAs are not completely failsafe. Virus levels within potato tubers, which are screened by ELISA for use as seed potatoes, are normally low while the tubers are dormant. ELISA detection of viruses in these potatoes is difficult and absorbance values may fall below the set cut-off value. For this reason, seed tuber screening is performed on sprouting rather than dormant tubers. Although this results in more reliable readings than direct tuber testing, it does delay the certification of seed potatoes. Another disadvantage of an immuno-based detection method is that changes at the gene level may have an influence on the immunogenicity of the antigen to be detected. In terms of potato plant viruses, mutations within the CP gene may cause the CP to undergo conformational changes rendering antibodies produced against the previously present virus less effective. RT-PCR Reverse transcriptase polymerase chain reaction (RT-PCR) has become a powerful and effective method for detection of potato plant viruses within potato plant material and even dormant potatoes. Only a minute piece of plant material is required for analysis using RT-PCR. Considering the protocol described within this thesis, 0.1 g of plant material is enough for 14 500 separate reactions. During a RT-PCR specific target RNA sequences are amplified exponentially into DNA copies. For this to occur, however, the RNA of the virus must first be transcribed to DNA by means of a reverse transcriptase polymerase. This polymerase synthesizes a DNA strand using the RNA as template. This results in a DNA/RNA complex. For synthesis of a DNA strand from the RNA template only the reverse primer is required since the RNA is a single strand arranged from 5’ to 3’. Subsequently, the newly synthesized DNA strand is used as a template for traditional PCR. Different types of reverse transcriptase polymerases are available to suit different needs and reaction conditions. Reverse transcriptase enzymes commonly used include AMV RT, SuperScript III, ImProm-II, Omniscript, Sensiscript and Tth RT. At the end of the RT step the polymerase enzyme is heatactivated. It could also be that the reverse transcriptase polymerase and DNA polymerase is one and the same enzyme and that the enzyme only requires a DNA polymerase activation step after the RT step. An example of such an enzyme is Tth polymerase. This enzyme has both RNA-dependent reverse transcriptase and DNA-dependent polymerase activity. However, the active center of the DNA polymerase is covered by dedicated oligonucleotides, called aptamers. At temperatures below the optimal reaction temperature of the DNA-dependent polymerase component of Tth remains covered by the aptamers. At these temperatures the Tth enzyme only synthesizes a DNA copy of the RNA template. Once the reaction temperature is raised to 95 °C, the aptamers are removed and the DNA-dependent polymerase component will start to amplify the target sequence. PCR amplification of the DNA target occurs in three steps: denaturation, annealing and extension. RT-PCR also allows for the detection of several viral targets in the same reaction through the use of several primer combinations. This is called multiplexing. Although multiplexing is technically more demanding than a traditional simplex reaction it allows for a higher throughput in that a single sample can be tested for several viral strains in a single reaction. Primers used for multiplexing are chosen in such a manner that they result in amplicons of various sizes. This allows for post RT-PCR analysis using gel electrophoresis. Although RT-PCR saves time, allows for multiplexing and is more sensitive than ELISA, the reagents and instrumentation needed are expensive and require a higher level of technical expertise. Also, end product analysis using gel electrophoresis is laborious, relatively more expensive, time-consuming and does not lend itself to automation. For these reasons the use of RT-PCR for routine screening is not feasible and has not replaced ELISA. It does, however, provide the industry with the opportunity to screen borderline cases, especially in the case of seed potato certification. Quantitative PCR In most traditional PCRs the resulting products are analyzed after the PCR has been completed. This is called end-point analysis and is normally qualitative of nature rather than being quantitative. For this sort of analysis, products are mostly analyzed on an agarose gel and visualized using ethidium bromide as a fluorescent dye. Direct correlation between signal strength and initial sample concentration is not possible using end-point analysis since PCR efficiency decreases as the reaction nears the plateau phase. Quantitative PCR, however, offers an accurate and rapid alternative to traditional PCR. Quantitative PCR offers the researcher the opportunity to amplify and analyze the product in a single tube using fluorescent dyes. This is known as homogeneous PCR. During a quantitative PCR the increase in fluorescence is correlated with the increase in product. Through the use of different specific, dyes quantitative PCR can be used to distinguish between different strains of a virus and even to detect point mutations. The major advantage of quantitative PCR is that analysis of resulting products using gel electrophoresis is not required. This means that quantitative PCR can be implemented as a high-throughput technique for sample screening. Quantitative PCR has been described for detection and discrimination of PVYO and PVYN isolates and for reliable discrimination between PVYNTN and PVYN isolates. == See also ==
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